
One of the most common concerns among patients in St. Louis is:
“My skin feels loose. It doesn’t feel firm anymore.”
Skin laxity is a natural biological process, not a flaw. It affects the face, neck, abdomen, arms, and almost any area of the body. Understanding it requires understanding the structure, architecture, and physiology of the skin itself.
This blog explores the science of skin laxity, including collagen decline, elastin breakdown, inflammation, genetics, sun exposure, and how aging affects the skin’s deeper support layers.
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Skin is made of several layers:
Skin laxity develops when the deeper layers weaken.
Collagen gives skin its firmness. Beginning in the mid-20s, collagen decreases 1–2% per year.
Elastin allows skin to snap back after being stretched. It is extremely slow to regenerate.
With age, superficial and deep fat pads shift, contributing to skin looseness.
Underlying muscles weaken or atrophy with age.
Facial bone structure changes with age, creating the appearance of sagging even without skin changes.
Inflammation breaks down collagen and elastin at an accelerated rate.
Sun exposure is the #1 external cause of skin laxity.
Genetic patterns influence collagen density, oil production, and structural support.
Estrogen and testosterone both support collagen production.
Skin aging is influenced by:
Aging skin produces less collagen and repairs itself more slowly.
Patients often describe:
Loose skin is not vanity — it is an understandable response to normal aging.
During evaluations, we assess:
Understanding these variables guides personalized education and expectations.
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Factors that accelerate laxity include:
Skin laxity is a natural part of aging, but understanding the physiology behind it empowers patients to make informed choices about their skin and health.
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Learn more about skin health and aging in our St. Louis clinic.